The adenoviral E1A oncoprotein activates the Smad7 promoter: requirement of a functional E-box

Int J Oncol. 2009 Dec;35(6):1493-8. doi: 10.3892/ijo_00000468.

Abstract

DNA tumorviruses like adenoviruses (AdV) or human papillomaviruses (HPV) have adopted various strategies to interfere with antiproliferative transforming growth factor-beta (TGF-beta) signalling. Here we report that the AdV E1A oncoprotein is sufficient to induce Smad7 expression, an inhibitor of TGF-beta signalling. E1A but not HPV oncoproteins activated the Smad7 promoter. A promoter proximal E-box was crucial for E1A-mediated transcriptional activity. E1A but not HPV oncoproteins induced specific binding activity at this E-box, which was identified as upstream stimulatory factor. In conclusion, these results unravel a novel mechanism of how the AdV E1A oncoprotein induces a cellular inhibitor of TGF-beta signalling.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenovirus E1A Proteins / metabolism*
  • Cell Line, Tumor
  • Cell Transformation, Viral / physiology*
  • E-Box Elements / genetics*
  • Electrophoretic Mobility Shift Assay
  • Humans
  • Papillomaviridae
  • Promoter Regions, Genetic / genetics*
  • Reverse Transcriptase Polymerase Chain Reaction
  • Signal Transduction / physiology
  • Smad7 Protein / genetics*
  • Smad7 Protein / metabolism
  • Transfection
  • Transforming Growth Factor beta / metabolism

Substances

  • Adenovirus E1A Proteins
  • SMAD7 protein, human
  • Smad7 Protein
  • Transforming Growth Factor beta