Sertindole improves sub-chronic PCP-induced reversal learning and episodic memory deficits in rodents: involvement of 5-HT(6) and 5-HT (2A) receptor mechanisms

Psychopharmacology (Berl). 2010 Jan;208(1):23-36. doi: 10.1007/s00213-009-1702-5. Epub 2009 Oct 23.

Abstract

Aim: This study examined the efficacy of sertindole in comparison with a selective 5-HT(6) and a 5-HT(2A) receptor antagonist to reverse sub-chronic phencyclidine (PCP)-induced cognitive deficits in female rats.

Methods: In the first test, adult female hooded Lister rats were trained to perform an operant reversal learning task to 90% criterion. After training, rats were treated with PCP at 2 mg/kg (i.p.) or vehicle twice daily for 7 days, followed by 7 days washout. For the second test, novel object recognition (NOR), a separate batch of rats, had the same sub-chronic PCP dosing regime and washout period. In reversal learning, rats were treated acutely with sertindole, the selective 5-HT(2A) receptor antagonist M100.907 or the selective 5-HT(6) receptor antagonist SB-742457.

Results: The PCP-induced selective reversal learning deficit was significantly improved by sertindole, M100.907 and SB-742457. Sertindole also significantly improved the sub-chronic PCP-induced deficit in NOR, a test of episodic memory following a 1 min and 1 h inter-trial interval. In vivo binding studies showed that the dose-response relationship for sertindole in this study most closely correlates with affinity for 5-HT(6) receptor in vivo binding in striatum, although contribution from binding to 5-HT(2A) receptors in vivo in cortex may also provide an important mechanism.

Conclusion: The efficacies of selective 5-HT(2A) and 5-HT(6) receptor antagonists suggest potential mechanisms mediating the effects of sertindole, which has high affinity for these 5-HT receptor subtypes. The sertindole-induced improvement in cognitive function in this animal model suggests relevance for the management of cognitive deficit symptoms in schizophrenia.

Publication types

  • Comparative Study

MeSH terms

  • Animals
  • Antipsychotic Agents / administration & dosage
  • Antipsychotic Agents / pharmacology*
  • Cognition Disorders / chemically induced
  • Cognition Disorders / drug therapy*
  • Disease Models, Animal
  • Dose-Response Relationship, Drug
  • Female
  • Fluorobenzenes / pharmacology
  • Imidazoles / administration & dosage
  • Imidazoles / pharmacology*
  • Indoles / administration & dosage
  • Indoles / pharmacology*
  • Memory Disorders / chemically induced
  • Memory Disorders / drug therapy
  • Phencyclidine / toxicity*
  • Piperidines / pharmacology
  • Quinolines / pharmacology
  • Rats
  • Receptor, Serotonin, 5-HT2A / metabolism
  • Receptors, Serotonin / metabolism
  • Reversal Learning / drug effects
  • Schizophrenia / drug therapy
  • Schizophrenia / physiopathology
  • Sulfones / pharmacology

Substances

  • 3-benzenesulfonyl-8-piperazin-1-ylquinoline
  • Antipsychotic Agents
  • Fluorobenzenes
  • Imidazoles
  • Indoles
  • Piperidines
  • Quinolines
  • Receptor, Serotonin, 5-HT2A
  • Receptors, Serotonin
  • Sulfones
  • serotonin 6 receptor
  • volinanserin
  • sertindole
  • Phencyclidine