Abstract
The p53 target gene Wig-1 encodes a double-stranded-RNA-binding zinc finger protein. We show here that Wig-1 binds to p53 mRNA and stabilizes it through an AU-rich element (ARE) in the 3' UTR of the p53 mRNA. This effect is mirrored by enhanced p53 protein levels in both unstressed cells and cells exposed to p53-activating stress agents. Thus, the p53 target Wig-1 is a previously undescribed ARE-regulating protein that acts as a positive feedback regulator of p53, with implications both for the steady-state levels of p53 and for the p53 stress response. Our data reveal a previously undescribed link between the tumor suppressor p53 and posttranscriptional gene regulation via AREs in mRNA.
Publication types
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Research Support, Non-U.S. Gov't
MeSH terms
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3' Untranslated Regions
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Animals
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Base Composition
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Carrier Proteins / antagonists & inhibitors
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Carrier Proteins / genetics
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Carrier Proteins / metabolism*
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Cell Line
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Cell Nucleus / metabolism
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Cytoplasm / metabolism
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Feedback, Physiological
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Genes, p53
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Humans
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Mice
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NIH 3T3 Cells
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Nuclear Proteins / antagonists & inhibitors
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Nuclear Proteins / genetics
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Nuclear Proteins / metabolism*
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RNA Stability*
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RNA, Messenger / chemistry
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RNA, Messenger / genetics*
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RNA, Messenger / metabolism*
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RNA, Small Interfering / genetics
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RNA-Binding Proteins
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Stress, Physiological
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Tumor Suppressor Protein p53 / metabolism*
Substances
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3' Untranslated Regions
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Carrier Proteins
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Nuclear Proteins
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RNA, Messenger
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RNA, Small Interfering
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RNA-Binding Proteins
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TP53 protein, human
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Tumor Suppressor Protein p53
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ZMAT3 protein, human