Abstract
Lipoprotein cholesterol is mobilized from lysosomes by actions of the NPC1 and NPC2 proteins. In this issue of Cell Metabolism, Harrison et al. report on identification of an NPC2-interacting protein, the Nogo-B receptor, that regulates NPC2 protein levels. NPC2 stabilization may represent a novel mechanism through which cells respond to endocytosed cholesterol.
MeSH terms
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Carrier Proteins / metabolism*
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Cholesterol / metabolism
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Glycoproteins / metabolism*
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Humans
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RNA, Small Interfering / metabolism
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Receptors, Cell Surface / genetics
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Receptors, Cell Surface / metabolism
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Vesicular Transport Proteins
Substances
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Carrier Proteins
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Glycoproteins
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NPC2 protein, human
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NUS1 protein, human
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RNA, Small Interfering
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Receptors, Cell Surface
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Vesicular Transport Proteins
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Cholesterol