Decrease of Lp(a) during weight reduction in obese children is modified by the apo(a) kringle-IV copy number variation

Int J Obes (Lond). 2009 Oct;33(10):1136-42. doi: 10.1038/ijo.2009.144. Epub 2009 Jul 28.

Abstract

Background: Lipoprotein(a) [Lp(a)] is considered an independent risk factor for cardiovascular disease. Its concentration is mainly determined by the kringle-IV repeat copy number variation (CNV) at the apolipoprotein(a) [apo(a)] locus.

Objective: We aimed to investigate the immediate effect of weight reduction on plasma Lp(a) levels and its dependency on the apo(a) CNV in obese children.

Design: We performed a prospective longitudinal intervention study of a low-fat hypocaloric diet conducted in a 3-week dietary camp for obese children. In all, 140 obese participants (54 boys and 86 girls) with a mean age of 12.5+/-1.6 years and a mean relative body mass index (BMI) before treatment of 165.6+/-24.7% were included. Body weight and plasma levels of Lp(a), lipids, apolipoproteins A-I and B, insulin, and C-reactive protein were determined before the onset and after the end of the intervention. In addition, the number of apo(a) kringle-IV repeats were determined using sodium dodecyl sulfate agarose gel electrophoresis.

Results: The mean loss of body weight was 5.0+/-1.3 kg (-6.6%), resulting in a mean decrease of the relative BMI of 6.6%. Blood chemistry revealed significant changes in all parameters, especially in Lp(a), with a decrease from 24.4+/-30.6 to 17.9+/-22.6 mg per 100 ml or -19% (P<0.001). The decrease of Lp(a) levels was higher in the group with low compared with high molecular weight apo(a) phenotypes (-23.9 vs -16.6%).

Conclusions: Weight reduction in obese children is associated with significant changes in Lp(a) levels, especially in subjects with high pre-treatment Lp(a) concentrations. This effect is markedly influenced by the molecular phenotype at the copy-number variable apo(a) locus.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Apolipoproteins A / blood*
  • Apolipoproteins A / genetics
  • Body Mass Index
  • Cardiovascular Diseases / blood*
  • Cardiovascular Diseases / genetics
  • Child
  • Diet, Fat-Restricted
  • Female
  • Humans
  • Kringles / genetics
  • Kringles / physiology*
  • Lipoprotein(a) / blood*
  • Lipoprotein(a) / genetics
  • Longitudinal Studies
  • Male
  • Obesity / blood*
  • Obesity / genetics
  • Phenotype
  • Prospective Studies
  • Risk Factors
  • Weight Loss / genetics
  • Weight Loss / physiology*

Substances

  • Apolipoproteins A
  • Lipoprotein(a)