Increased 12-HETE production in bovine lymphocytes during selenium deficiency

Biochem Biophys Res Commun. 1991 Nov 27;181(1):389-95. doi: 10.1016/s0006-291x(05)81431-7.

Abstract

When peripheral blood lymphocytes were incubated with arachidonic acid in the presence of Ca++ ionophore (A23187), the cells from the selenium-deficient dairy cows produced significantly greater quantities of 12-hydroxyeicosatetraenoic acid (12-HETE) than the cells from the selenium-supplemented animals. The major product of reaction was verified as 12-HETE by cochromatography with a 12-HETE standard on an HPLC and structural analysis by GC-MS. Additionally, concanavalin A-stimulated lymphocyte proliferation was significantly decreased in cells from the Se-deficient cows. Furthermore, 12-HETE generated by the A23187-stimulated lymphocytes inhibited lymphocyte proliferation when added to Se-supplemented cell cultures. These observations suggest that self-regulation of lymphocyte proliferation might be mediated by 12-HETE production, especially during an altered nutritional state such as Se deficiency.

MeSH terms

  • 12-Hydroxy-5,8,10,14-eicosatetraenoic Acid
  • Animals
  • Arachidonic Acids / metabolism
  • Calcimycin / pharmacology
  • Cattle
  • Female
  • Hydroxyeicosatetraenoic Acids / biosynthesis*
  • Hydroxyeicosatetraenoic Acids / isolation & purification
  • Lipoxygenase / metabolism
  • Lymphocyte Activation
  • Lymphocytes / drug effects
  • Lymphocytes / immunology
  • Lymphocytes / physiology*
  • Mass Spectrometry
  • Selenium / deficiency*
  • Selenium / pharmacology

Substances

  • Arachidonic Acids
  • Hydroxyeicosatetraenoic Acids
  • Calcimycin
  • 12-Hydroxy-5,8,10,14-eicosatetraenoic Acid
  • Lipoxygenase
  • Selenium