Microcystins -LA, -YR, and -LR action on neutrophil migration

Biochem Biophys Res Commun. 2009 Apr 24;382(1):9-14. doi: 10.1016/j.bbrc.2009.02.009.

Abstract

Microcystins (MCs) produced by some freshwater cyanobacterial species possess potent liver toxicity as evidenced by acute neutrophil infiltration. Here, we investigate the ability of three structurally distinct toxins (MC-LA, MC-LR, and MC-YR) to evoke neutrophil recruitment per se and their effects on migration pathways. Intravital microscopic studies showed that topical application of only MC-LR enhanced the numbers of rolling and adhered leukocytes in the endothelium of postcapillary mesenteric venules. The latter effects may be dependent upon induction of the synthesis and expression of L-selectin and beta2-integrin in neutrophils, as assessed by flow cytometry and RT-PCR, respectively. Conversely, the three toxins promoted direct locomotion of neutrophils and enhanced their migration in response to fMLP, as measured by Boyden chamber assays, and increased intracellular calcium, a messenger in the chemotaxic process. In conclusion, our results show that MCs act on specific pathways of neutrophil recruitment, indicating their potential effect on neutrophils activation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Calcium / metabolism
  • Cell Adhesion
  • Leukocyte Rolling*
  • Male
  • Marine Toxins
  • Microcystins / pharmacology*
  • Neutrophil Infiltration*
  • Neutrophils / drug effects*
  • Neutrophils / immunology
  • Rats
  • Rats, Wistar

Substances

  • Marine Toxins
  • Microcystins
  • microcystin YR
  • cyanoginosin-LA
  • cyanoginosin LR
  • Calcium