Contribution of human herpesvirus 6 (HHV-6) viral load in whole blood and serum to investigate integrated HHV-6 transmission after haematopoietic stem cell transplantation

J Clin Virol. 2009 May;45(1):43-6. doi: 10.1016/j.jcv.2009.02.006. Epub 2009 Mar 24.

Abstract

Background: Human herpesvirus 6 (HHV-6) is susceptible to latency and recurrence. A less-frequent form of HHV-6 persistence is the integration of viral DNA into host chromosomes.

Objectives: To investigate HHV-6 viral load after haematopoietic stem cell transplantation (HSCT) in whole blood (WB) and serum with regard to integrated HHV-6 transmission diagnosis.

Study design: HHV-6 DNA quantitation in serum and WB was performed using quantitative polymerase chain reaction for the follow-up of a 16-year-old girl after HSCT. In whole blood, results were expressed as HHV-6 genomic equivalent copies (gec) per milliliter of WB or per million cells.

Results: HHV-6 viral load (undetectable before HSCT) increased up to 3.05 x 10(7)gec/10(6)cells. HHV-6 viral load in the donor sample (3.44 x 10(6)gec/10(6)cells) was in favor of viral transmission through HSCT. The correlation between viral load in WB and serum was significant (p=0.0005). Viral load results expressed as gec/10(6)cells in WB was more reliable than results expressed as gec/ml of whole blood.

Conclusion: These findings indicate that HHV-6 may be transmitted during HSCT as integrated virus contained in the graft. This reiterates that in the setting of HSCT, HHV-6 viral load must be correctly interpreted. Using HHV-6 viral load expressed as gec/10(6) cells may be more suitable for the follow-up of patients with integrated HHV-6.

MeSH terms

  • Adolescent
  • Adult
  • Analysis of Variance
  • Antiviral Agents / pharmacology
  • DNA, Viral / blood*
  • Female
  • Hematopoietic Stem Cell Transplantation*
  • Herpesvirus 6, Human / genetics*
  • Herpesvirus 6, Human / physiology
  • Humans
  • Male
  • Polymerase Chain Reaction
  • Regression Analysis
  • Roseolovirus Infections / transmission*
  • Tissue Donors*
  • Viral Load
  • Virus Integration*
  • Virus Replication / drug effects

Substances

  • Antiviral Agents
  • DNA, Viral