Impaired myelopoiesis in mice lacking the repressors of translation initiation, 4E-BP1 and 4E-BP2

Immunology. 2009 Sep;128(1 Suppl):e376-84. doi: 10.1111/j.1365-2567.2008.02981.x. Epub 2008 Oct 31.

Abstract

We investigated the role of two repressors of translation initiation in granulocytic differentiation using mice with a null mutation in the 4E-BP1 gene or with a null mutation in the 4E-BP2 gene. We show that 4E-BP1(-/-) and 4E-BP2(-/-) mice exhibit an increased number of immature granulocytic precursors, associated with a decreased number of mature granulocytic elements compared with wild-type mice, which is suggestive of an impaired granulocytic differentiation. Clonogenetic analyses revealed a reduced number of granulocytic colonies and concomitant increase in granulo-monocytic colonies in 4E-BP(-/-) mice. Finally, a slight expansion of monocytic cells was observed in the 4E-BP2(-/-) mice. In contrast, we did not observe any significant difference in thymocyte maturation in these mice. These results, together with the fact that 4E-BPs are markedly induced during granulo-monocytic differentiation of myeloid cells in vitro, highlight the pivotal role of 4E-BP1 and 4E-BP2 in the early phases of myelopoiesis. These results represent the first in vivo evidence of the involvement of translation in the early phases of granulo-monocytic differentiation and further extend the role of translation in haematopoietic differentiation.

Publication types

  • Research Support, N.I.H., Extramural

MeSH terms

  • Adaptor Proteins, Signal Transducing
  • Animals
  • Bone Marrow / immunology
  • Bone Marrow / metabolism
  • Carrier Proteins / genetics
  • Carrier Proteins / immunology*
  • Cell Cycle Proteins
  • Cell Differentiation / immunology*
  • Eukaryotic Initiation Factors / genetics
  • Eukaryotic Initiation Factors / immunology*
  • Granulocytes / immunology*
  • Granulocytes / metabolism
  • Mice
  • Mice, Inbred BALB C
  • Mice, Knockout
  • Monocytes / immunology
  • Monocytes / metabolism
  • Myelopoiesis / immunology*
  • Phosphoproteins / genetics
  • Phosphoproteins / immunology*
  • Spleen / immunology
  • Spleen / metabolism
  • Stem Cells / immunology
  • Stem Cells / metabolism
  • T-Lymphocytes / immunology
  • T-Lymphocytes / metabolism
  • Thymus Gland / immunology
  • Thymus Gland / metabolism

Substances

  • Adaptor Proteins, Signal Transducing
  • Carrier Proteins
  • Cell Cycle Proteins
  • Eif4ebp1 protein, mouse
  • Eif4ebp2 protein, mouse
  • Eukaryotic Initiation Factors
  • Phosphoproteins