Induction of glutathione S-transferase P-form in primary cultured rat hepatocytes by epidermal growth factor and insulin

Jpn J Cancer Res. 1991 Jul;82(7):807-14. doi: 10.1111/j.1349-7006.1991.tb02706.x.

Abstract

The effects of epidermal growth factor (EGF) (10 ng/ml) and insulin (100 nM) on the expression of glutathione S-transferases (GSTs), especially the GST-P form (GST 7-7), were examined in primary cultured rat hepatocytes in serum-free medium. On culture with EGF and insulin, the GST activities towards 1-chloro-2,4-dinitrobenzene and 1,2-dichloro-4-nitrobenzene were transiently decreased on day 2 to 10% of those of freshly isolated hepatocytes and then increased to 60 to 100% of those of freshly isolated cells on day 4. Western blot analysis of GSTs revealed that GST-P, which is not present in freshly isolated hepatocytes, was markedly induced and that GST subunits 3 and 4 of the Mu class also increased after addition of EGF and/or insulin, while the subunits 1 and 2 of the Alpha class disappeared. Northern blot analysis showed that on addition of EGF and insulin the level of GST-P mRNA was also elevated and expressions of the nuclear oncogenes c-jun and c-fos were enhanced. These results suggest that the enhanced expression of GST-P induced by EGF or insulin in primary cultured rat hepatocytes might be regulated by JUN and FOS proteins.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cells, Cultured
  • DNA-Binding Proteins / genetics
  • Enzyme Induction
  • Epidermal Growth Factor / pharmacology*
  • Gene Expression / physiology
  • Glutathione Transferase / biosynthesis*
  • Glutathione Transferase / genetics
  • Glutathione Transferase / metabolism
  • Insulin / pharmacology*
  • Isoenzymes / biosynthesis*
  • Isoenzymes / genetics
  • Isoenzymes / metabolism
  • Liver / cytology
  • Liver / drug effects
  • Liver / enzymology*
  • Male
  • Proto-Oncogene Proteins / genetics
  • Proto-Oncogene Proteins c-fos
  • Proto-Oncogene Proteins c-jun
  • Rats
  • Rats, Inbred Strains
  • Tetradecanoylphorbol Acetate / pharmacology
  • Transcription Factors / genetics
  • Transcription, Genetic / drug effects
  • Transforming Growth Factor beta / pharmacology

Substances

  • DNA-Binding Proteins
  • Insulin
  • Isoenzymes
  • Proto-Oncogene Proteins
  • Proto-Oncogene Proteins c-fos
  • Proto-Oncogene Proteins c-jun
  • Transcription Factors
  • Transforming Growth Factor beta
  • Epidermal Growth Factor
  • Glutathione Transferase
  • Tetradecanoylphorbol Acetate