Molecular imaging of phosphorylation events for drug development

Mol Imaging Biol. 2009 May-Jun;11(3):144-58. doi: 10.1007/s11307-008-0187-7. Epub 2008 Dec 2.

Abstract

Purpose: Protein phosphorylation mediated by protein kinases controls numerous cellular processes. A genetically encoded, generalizable split firefly luciferase (FL)-assisted complementation system was developed for noninvasive monitoring phosphorylation events and efficacies of kinase inhibitors in cell culture and in small living subjects by optical bioluminescence imaging.

Procedures: An Akt sensor (AST) was constructed to monitor Akt phosphorylation and the effect of different PI-3K and Akt inhibitors. Specificity of AST was determined using a non-phosphorylable mutant sensor containing an alanine substitution (ASA).

Results: The PI-3K inhibitor LY294002 and Akt kinase inhibitor perifosine led to temporal- and dose-dependent increases in complemented FL activities in 293T human kidney cancer cells stably expressing AST (293T/AST) but not in 293T/ASA cells. Inhibition of endogenous Akt phosphorylation and kinase activities by perifosine also correlated with increase in complemented FL activities in 293T/AST cells but not in 293T/ASA cells. Treatment of nude mice bearing 293T/AST xenografts with perifosine led to a 2-fold increase in complemented FL activities compared to that of 293T/ASA xenografts. Our system was used to screen a small chemical library for novel modulators of Akt kinase activity.

Conclusion: This generalizable approach for noninvasive monitoring of phosphorylation events will accelerate the discovery and validation of novel kinase inhibitors and modulators of phosphorylation events.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Line
  • Drug Discovery / methods*
  • Genetic Complementation Test
  • Humans
  • Luciferases, Firefly / genetics
  • Luminescence
  • Mice
  • Molecular Probe Techniques*
  • Molecular Probes
  • Mutation
  • Phosphatidylinositol 3-Kinases / metabolism
  • Phosphorylation* / drug effects
  • Protein Kinase Inhibitors / pharmacology*
  • Proteins / metabolism*
  • Proto-Oncogene Proteins c-akt / genetics
  • Proto-Oncogene Proteins c-akt / metabolism*

Substances

  • Molecular Probes
  • Protein Kinase Inhibitors
  • Proteins
  • Luciferases, Firefly
  • Phosphatidylinositol 3-Kinases
  • Proto-Oncogene Proteins c-akt