Serum zinc in the progression of Alzheimer's disease

J Alzheimers Dis. 2008 Nov;15(3):443-50. doi: 10.3233/jad-2008-15310.

Abstract

Previous studies show significantly decreased levels of zinc transporter 1 (ZnT-1) in the brain of subjects with mild cognitive impairment (MCI) but significantly increased ZnT-1 in late stage AD (LAD). However, the reason for the apparent dichotomy is unclear. Based on in vivo studies that show animals provided a zinc (Zn) deficient diet demonstrate decreased brain ZnT-1, we used inductively coupled plasma-mass spectrometry (ICP-MS) to quantify serum Zn levels from 18 living mild to moderate AD patients (9 men, 9 women), 19 MCI patients (9 men, 10 women) and 16 age-matched normal control (NC) subjects (9 men, 7 women). Zinc levels for all subjects were not significantly different among any of the three subject groups. However, there was a statistically significant decrease of serum Zn (11.7 +/- 0.5 microM) in men with MCI compared to women with MCI (13.7 +/- 0.6 microM) and NC men (13.9 +/- 0.6 microM). Serum Zn levels in probable AD patients were comparable to those in NC subjects. Overall, these data suggest a significant decrease of serum Zn in men with MCI, may explain the loss of ZnT-1 observed in previous studies and suggest there may be more pronounced sex differences in MCI than were previously recognized.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Alzheimer Disease / blood*
  • Apolipoprotein E4 / genetics
  • Cation Transport Proteins / metabolism
  • Disease Progression
  • Female
  • Humans
  • Male
  • Mass Spectrometry
  • Neuropsychological Tests
  • Reproducibility of Results
  • Sex Characteristics
  • Zinc / blood*

Substances

  • Apolipoprotein E4
  • Cation Transport Proteins
  • SLC30A1 protein, human
  • Zinc