Abstract
Alzheimer disease treatment has yet to yield a successful therapy that addresses the source of the damage found in brains. Of the varied proposed theories of AD etiology, reactive oxygen species (ROS) generation is cited as a common factor. Efforts to reduce the pathology associated with ROS via antioxidants therefore offer new hope to patients suffering from this devastative disease.
MeSH terms
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Alzheimer Disease / drug therapy*
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Alzheimer Disease / genetics
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Alzheimer Disease / metabolism
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Alzheimer Disease / pathology
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Animals
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Antioxidants / pharmacology
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Antioxidants / therapeutic use*
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Brain / pathology
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Free Radical Scavengers / pharmacology
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Free Radical Scavengers / therapeutic use
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Humans
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Oxidative Stress / drug effects
Substances
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Antioxidants
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Free Radical Scavengers