Reconfiguration of genomic anchors upon transcriptional activation of the human major histocompatibility complex

Genome Res. 2008 Nov;18(11):1778-86. doi: 10.1101/gr.082313.108. Epub 2008 Oct 10.

Abstract

The folding of chromatin into topologically constrained loop domains is essential for genomic function. We have identified genomic anchors that define the organization of chromatin loop domains across the human major histocompatibility complex (MHC). This locus contains critical genes for immunity and is associated with more diseases than any other region of the genome. Classical MHC genes are expressed in a cell type-specific pattern and can be induced by cytokines such as interferon-gamma (IFNG). Transcriptional activation of the MHC was associated with a reconfiguration of chromatin architecture resulting from the formation of additional genomic anchors. These findings suggest that the dynamic arrangement of genomic anchors and loops plays a role in transcriptional regulation.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • B-Lymphocytes / drug effects
  • B-Lymphocytes / immunology
  • Cell Line
  • Chromatin / chemistry
  • Chromatin / genetics
  • Fibroblasts / drug effects
  • Fibroblasts / immunology
  • Genes, MHC Class II
  • Genome, Human
  • HLA Antigens / genetics
  • Humans
  • Interferon-gamma / pharmacology
  • Major Histocompatibility Complex*
  • Matrix Attachment Regions / drug effects
  • Models, Genetic
  • Recombinant Proteins
  • Transcriptional Activation

Substances

  • Chromatin
  • HLA Antigens
  • Recombinant Proteins
  • Interferon-gamma