The macrophage in tuberculosis: sinner or saint? The T cell decides

Pathobiology. 1991;59(3):153-5. doi: 10.1159/000163634.

Abstract

Immunity to intracellular bacteria including Mycobacterium tuberculosis crucially depends on intricate interactions between T lymphocytes and macrophages. Before contact with T lymphocytes macrophages serve as habitat for M. tuberculosis organisms; after activation by T cells they become major effectors against these pathogens. T cells comprise different subsets which express different functional activities. This paper describes evidence that different T cell subsets (CD4 alpha/beta T cells, CD8 alpha/beta T cells and gamma/delta T cells) as well as different T cell functions (interleukin secretion and target cell lysis) contribute to immunity against tuberculosis.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Humans
  • Interleukins / immunology
  • Leprosy / immunology
  • Macrophages / immunology*
  • Receptors, Antigen, T-Cell
  • T-Lymphocyte Subsets / immunology
  • Tuberculosis / immunology*

Substances

  • Interleukins
  • Receptors, Antigen, T-Cell