The pathogenesis of gout

Cleve Clin J Med. 2008 Jul:75 Suppl 5:S2-4. doi: 10.3949/ccjm.75.suppl_5.s2.

Abstract

An elevated serum urate level, together with local factors, can result in the deposition of urate crystals into the joints. Once crystals are deposited into a joint, they can be released into the joint space and initiate an inflammatory cascade causing acute gouty arthritis. These acute flares resolve, but the crystals remain in the joint. The way to ultimately correct the underlying metabolic problem of hyperuricemia and the crystal deposition is to lower the serum urate level and dissolve the crystal deposits. This will stop both the acute attacks and the progressive joint damage.

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Gout / blood
  • Gout / etiology*
  • Gout / pathology
  • Humans
  • Prognosis
  • Risk Factors
  • Synovial Membrane / pathology
  • Uric Acid / blood

Substances

  • Uric Acid