Enhanced glycolysis induced by mtDNA mutations does not regulate metastasis

FEBS Lett. 2008 Oct 15;582(23-24):3525-30. doi: 10.1016/j.febslet.2008.09.024. Epub 2008 Sep 19.

Abstract

We addressed the issue of whether enhanced glycolysis caused by mtDNA mutations independently induces metastasis in tumor cells using mtDNA transfer technology. The resultant trans-mitochondrial cybrids sharing the same nuclear background of poorly metastatic carcinoma P29 cells, P29mtA11 and P29mtDelta cybrids, possessed mtDNA with a G13997A mutation from highly metastatic carcinoma A11 cells and mtDNA with a 4696bp deletion mutation, respectively. The P29mtDelta cybrids expressed enhanced glycolysis, but did not express ROS overproduction and high metastatic potential, whereas P29mtA11 cybrids showed enhanced glycolysis, ROS overproduction, and high metastatic potential. Thus, enhanced glycolysis alone does not induce metastasis in the cybrids.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Line, Tumor
  • Cell Nucleus / metabolism
  • Cell Respiration / genetics
  • DNA, Mitochondrial / genetics*
  • Genotype
  • Glycolysis / genetics*
  • Mice
  • Mitochondria / genetics
  • Mitochondria / metabolism*
  • Mutation
  • Myeloid Cell Leukemia Sequence 1 Protein
  • Neoplasm Metastasis / genetics*
  • Proto-Oncogene Proteins c-bcl-2 / biosynthesis
  • Reactive Oxygen Species / metabolism
  • Transfection

Substances

  • DNA, Mitochondrial
  • Myeloid Cell Leukemia Sequence 1 Protein
  • Proto-Oncogene Proteins c-bcl-2
  • Reactive Oxygen Species