Human amnion epithelial cells can be induced to differentiate into functional insulin-producing cells

Acta Biochim Biophys Sin (Shanghai). 2008 Sep;40(9):830-9.

Abstract

Pancreatic islet transplantation has demonstrated that long-term insulin independence may be achieved in patients suffering from diabetes mellitus type 1. However, limited availability of islet tissue means that new sources of insulin-producing cells that are responsive to glucose are required. Here, we show that human amnion epithelial cells (HAEC) can be induced to differentiate into functional insulin-producing cells in vitro. After induction of differentiation, HAEC expressed multiple pancreatic beta-cell genes, including insulin, pancreas duodenum homeobox-1, paired box gene 6, NK2 transcription factor-related locus 2, Islet 1, glucokinase, and glucose transporter-2, and released C-peptide in a glucose-regulated manner in response to other extracellular stimulations. The transplantation of induced HAEC into streptozotocin-induced diabetic C57 mice reversed hyperglycemia, restored body weight, and maintained euglycemia for 30 d. These findings indicated that HAEC may be a new source for cell replacement therapy in type 1 diabetes.

MeSH terms

  • Amnion / metabolism*
  • Amnion / surgery
  • Amnion / transplantation
  • Animals
  • C-Peptide / metabolism
  • Cell Differentiation / drug effects*
  • Cells, Cultured
  • Culture Media, Serum-Free
  • Diabetes Mellitus, Experimental / metabolism
  • Diabetes Mellitus, Experimental / surgery
  • Diabetes Mellitus, Type 1 / therapy
  • Epithelial Cells / metabolism*
  • Epithelial Cells / transplantation
  • Glucose / metabolism
  • Glucose / pharmacology*
  • Humans
  • Hyperglycemia / metabolism
  • Hyperglycemia / surgery
  • Hyperglycemia / therapy
  • Insulin-Secreting Cells / metabolism*
  • Insulin-Secreting Cells / transplantation
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Radioimmunoassay
  • Time Factors

Substances

  • C-Peptide
  • Culture Media, Serum-Free
  • Glucose