[Effects of acrylonitrile on the activation of nuclear factor-kappaB signaling pathways and related gene expression in rat brain and rat glial cells]

Wei Sheng Yan Jiu. 2008 May;37(3):264-8.
[Article in Chinese]

Abstract

Objective: To study the effects of acrylonitrile on the activation of nuclear factor kappaB (NF-kappaB) signaling pathways and related gene expressions in rat brain and rat glial cells.

Methods: The microarray and EMSA (electrophoretic mobility shift assay) techniques were used to investigated NF-kappaB pathway and related gene expressions changes in rat brain after treated with 0 mg/kg and 200mg/kg ACN for 14,28 and 90 days and rat normal glial cells after treated with 0, 25, 50 and 75 microg/ml ACN for 4 and 24 hours.

Results: ACN induced the NF-kappaB pathway related gene expression (NIK, IKK, NF-kappaB1, NF-kappaB 2, IkappaBa, c-myc) in rat brain (in vivo) and in rat glial cells (in vitro). It was confirmed by EMSA results that ACN caused activation of the NF-kappaB pathway in rat brain and rat glial cells.

Conclusion: There is some relationship between gene expression of NF-kappaB pathway and ACN-induced oxidative stress.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acrylonitrile / toxicity*
  • Animals
  • Brain / cytology
  • Brain / metabolism*
  • Cell Line
  • Male
  • NF-kappa B / genetics
  • NF-kappa B / metabolism*
  • Neuroglia / cytology
  • Neuroglia / drug effects
  • Neuroglia / metabolism*
  • Oxidative Stress / drug effects
  • Rats
  • Rats, Sprague-Dawley
  • Signal Transduction / drug effects*

Substances

  • NF-kappa B
  • Acrylonitrile