Abstract
Hepcidin, the systemic regulator of iron homeostasis is activated by proteins responsible for hereditary hemochromatosis, bone morphogenetic proteins (BMPs), and inflammatory cytokines. Three recent publications now identify a novel hepcidin suppressor, the transmembrane serine protease TMPRSS6 (also known as matriptase-2), which is required to sense iron deficiency.
Publication types
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Research Support, Non-U.S. Gov't
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Review
MeSH terms
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Anemia / etiology
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Animals
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Antimicrobial Cationic Peptides / biosynthesis
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Antimicrobial Cationic Peptides / genetics*
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Antimicrobial Cationic Peptides / physiology
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Gene Expression Regulation
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Hepcidins
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Homeostasis
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Humans
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Iron / metabolism
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Membrane Proteins / physiology*
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Serine Endopeptidases / physiology*
Substances
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Antimicrobial Cationic Peptides
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HAMP protein, human
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Hepcidins
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Membrane Proteins
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Iron
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Serine Endopeptidases
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matriptase 2