K(+) currents fail to change in reactive retinal glial cells in a mouse model of glaucoma

Graefes Arch Clin Exp Ophthalmol. 2008 Sep;246(9):1249-54. doi: 10.1007/s00417-008-0872-x. Epub 2008 Jun 11.

Abstract

Purpose: To investigate the membrane physiology of Müller glial cells from retinae of DBA/2J mice (which develop ocular hypertension) and of C57BL/6 control mice of different ages.

Methods: Retinae were obtained at the ages of 3, 6, and 12 months from DBA/2J mice and from C57BL/6 controls. Immunohistochemistry was performed using antibodies against glial fibrillary acidic protein (GFAP). Whole-cell membrane currents, membrane potentials and capacitances were recorded from freshly isolated Müller cells.

Results: Strong immunostaining for GFAP was found in Müller cells of 12-month-old DBA/2J mice, whereas only astrocytes were immunopositive in C57BL/6 retinae. No significant alterations of membrane currents or potentials of Müller cells from DBA/2J mice as compared to controls were observed at any age; however, the membrane capacitance was increased in Müller cells from DBA/2J mice at the age of 6 months.

Conclusions: Although Müller cells of DBA/2J mice display some symptoms of reactive gliosis, the lack of significant alterations of the membrane physiology confirm previous data demonstrating that these cells undergo a nonproliferative gliosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Astrocytes / metabolism
  • Disease Models, Animal
  • Female
  • Fluorescent Antibody Technique, Indirect
  • Glaucoma / physiopathology*
  • Glial Fibrillary Acidic Protein
  • Gliosis / physiopathology*
  • Membrane Potentials / physiology*
  • Mice
  • Mice, Inbred C57BL
  • Mice, Inbred DBA
  • Microscopy, Fluorescence
  • Nerve Tissue Proteins / metabolism
  • Neuroglia / physiology*
  • Ocular Hypertension / physiopathology
  • Patch-Clamp Techniques
  • Potassium Channels / physiology*
  • Retina / physiopathology*
  • Retinal Degeneration / physiopathology*

Substances

  • Glial Fibrillary Acidic Protein
  • Nerve Tissue Proteins
  • Potassium Channels
  • glial fibrillary astrocytic protein, mouse