[Impact of all-trans retinoic acid on gene expression profile of glioblastoma cell line SHG-44]

Ai Zheng. 2008 May;27(5):482-90.
[Article in Chinese]

Abstract

Background & objective: Astrocytoma has the trend of malignant progression. Differentiation-inducing therapy can induce tumor differentiation and make tumor cells become less malignant or even normal. This study was to investigate the impact of all-trans retinoic acid (ATRA) on the gene expression profile of glioblastoma cell line SHG-44, and to provide basic data for further research on gene therapy for human astrocytoma.

Methods: After treatment of 10 micromol/L ATRA, total RNA was extracted from SHG-44 cells for reverse transcription-polymerase chain reaction, and cDNA product was marked with fluorochromes Cy3 and Cy5. The gene expression profiles of SHG-44 cells before and after treatment of ATRA were detected by chip hybridization to identify differentially expressed genes. Some differentially expressed genes were selected randomly for Northern blot analysis.

Results: Forty-two differentially expressed genes were found by cDNA microarray: 28 were up-regulated and 14 were down-regulated in ATRA-treated SHG-44 cells as compared with those in untreated SHG-44 cells. These genes were functionally classified into several groups as follow: apoptosis, cell mobility and metastasis, cell cycle and growth regulation, cytoskeleton, differentiation, metabolic pathway, oncogene, oxidative phosphorylation, receptors and signal transduction, ribosome, ubiquitin-proteasome system, growth factor and cytokine, and so on.

Conclusions: ATRA can result in the changes of gene expression profiles in SHG-44 cells. These differentially expressed genes may mediate the mechanism of ATRA-induced differentiation of SHG-44 cells, and regulate tumor progression.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antineoplastic Agents / pharmacology
  • Brain Neoplasms* / metabolism
  • Brain Neoplasms* / pathology
  • Cell Cycle / drug effects
  • Cell Line, Tumor
  • Cell Proliferation / drug effects
  • Gene Expression Profiling*
  • Gene Expression Regulation, Neoplastic
  • Glioblastoma* / metabolism
  • Glioblastoma* / pathology
  • Humans
  • Neoplasm Proteins / metabolism
  • Oligonucleotide Array Sequence Analysis*
  • Ribosomal Proteins / metabolism
  • Superoxide Dismutase / metabolism
  • Tretinoin / pharmacology*

Substances

  • Antineoplastic Agents
  • Neoplasm Proteins
  • RPL13 protein, human
  • Ribosomal Proteins
  • Tretinoin
  • Superoxide Dismutase
  • superoxide dismutase 2