Ovarian cancer risk is associated with a common variant in the promoter sequence of the mismatch repair gene MLH1

Gynecol Oncol. 2008 Jun;109(3):384-7. doi: 10.1016/j.ygyno.2007.11.046. Epub 2008 Apr 10.

Abstract

Objectives: Inherited mutations in the MLH1 gene are associated with a proportion of families with the hereditary non-polyposis colon cancer syndrome (HNPCC). The cardinal features of the syndrome are a predisposition to colon, endometrial and ovarian cancers. Recently, it has been shown that a non-coding polymorphic variant in MLH1 (G>A nt-93) predisposes to colon and endometrial cancer, but with much reduced penetrance. We sought to establish whether or not this polymorphic variant also predisposes to ovarian cancer.

Methods: We genotyped 899 women with invasive ovarian cancer and 931 controls for the G>A nt-93 variant.

Results: The presence of the variant was associated with a modest, but highly significant risk of ovarian cancer (OR=1.5; 95% CI 1.3-1.9; p=0.00005). The association was present in cancers of all histologies except clear cell, and in all ethnic groups.

Conclusions: The G>A nt-93 variant of the MLH1 gene is associated with an increased risk of invasive ovarian cancer.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adaptor Proteins, Signal Transducing / genetics*
  • Case-Control Studies
  • Ethnicity / genetics
  • Family Health
  • Female
  • Genetic Predisposition to Disease
  • Genotype
  • Humans
  • Middle Aged
  • MutL Protein Homolog 1
  • Nuclear Proteins / genetics*
  • Ontario / epidemiology
  • Ovarian Neoplasms / epidemiology
  • Ovarian Neoplasms / genetics*
  • Ovarian Neoplasms / pathology
  • Polymorphism, Single Nucleotide
  • Promoter Regions, Genetic

Substances

  • Adaptor Proteins, Signal Transducing
  • MLH1 protein, human
  • Nuclear Proteins
  • MutL Protein Homolog 1