Electrocardiographic biomarkers of ventricular repolarisation in a single family of short QT syndrome and the role of the Bazett correction formula

Am J Cardiol. 2008 Mar 15;101(6):855-60. doi: 10.1016/j.amjcard.2007.10.049. Epub 2008 Feb 21.

Abstract

The definition of the short QT syndrome (SQTS) is based on QT duration, but thorough QT- and T-wave evaluation has not been performed to date. To evaluate the influence of QT rate-correction formulas in SQTS diagnosis, 12-lead electrocardiograms (ECGs) were recorded in 27 subjects from a single family with SQTS. Based on QT duration corrected by Bazett formula (QTc), 4 men were considered to have SQTS (QTc <or=340 ms), and 16 were classified to have "suspected" SQTS (QT <or=320 ms and/or QTc <or=380 ms). Holter ECGs were recorded in the 4 patients with SQTS, 8 with suspected SQTS, and 62 healthy volunteers. We evaluated QT duration at each RR interval and calculated individual alpha coefficient of the QT/RR relation. The QT interval duration at the mean Holter heart rate was corrected using an individual approach and 5 "universal" correction formulas (Bazett, Fridericia, Sagie-Framingham, Hodges, Rautaharju). Patients with SQTS and controls had lower alpha coefficients of the QT/RR relation when compared with Bazett and Fridericia formulas. The QT rate-dependence was lower in patients with SQTS (alpha 0.146 +/- 0.070) when compared with control subjects (alpha 0.203 +/- 0.039, p <0.05). The universal correction-formulas consistently provided higher values of corrected QT interval duration at the mean Holter heart rate than a subject-specific correction formula (p <0.05), which induced false negative diagnoses. In conclusion, the Bazett correction formula is not appropriate for making a diagnosis of SQTS. The subject-specific correction formula may provide a better cut-off value definition for the diagnosis of SQTS.

MeSH terms

  • Adolescent
  • Adult
  • Algorithms*
  • Bradycardia / diagnosis*
  • Bradycardia / genetics
  • Bradycardia / physiopathology
  • Diagnosis, Differential
  • Electrocardiography / methods*
  • Family*
  • Female
  • Follow-Up Studies
  • Genetic Predisposition to Disease*
  • Heart Rate / physiology*
  • Humans
  • Male
  • Models, Theoretical*
  • Pedigree
  • Prognosis