T cells, IgE antibodies, cytokines and allergic inflammation

Allerg Immunol (Paris). 1991 Jun;23(6):239-43.

Abstract

T helper cell (Th) subsets producing interleukin 4 (IL-4) or gamma interferon (IFN) play a reciprocal role in the regulation of human IgE synthesis. Though necessary, IL-4 alone was insufficient. Patients with hyper-IgE had significantly lower proportions of T-cells that produce IFN and TNF, but not IL-2 or IL-4. In patients with helminth infections or atopy production of IgE and eosinophilia may result from expansion of Th-2-like cells. T cell clones (TCC) specific for allergens and Toxocara canis (TES) showed dichotomy in cytokine secretion. Non-atopic donors gave insufficient TCC for comparison. Activation of the fibroblasts of the respiratory mucosae induces production of interstitial proteins such as collagen III, V, fibronectin.

Publication types

  • Review

MeSH terms

  • Airway Resistance
  • Cytokines / physiology*
  • Humans
  • Hypersensitivity / physiopathology*
  • Immunity, Cellular
  • Immunoglobulin E / physiology*
  • Inflammation / immunology*
  • Interleukin-4 / biosynthesis
  • Mast Cells / physiology
  • T-Lymphocytes / physiology*
  • T-Lymphocytes, Helper-Inducer / immunology

Substances

  • Cytokines
  • Interleukin-4
  • Immunoglobulin E