Histamine is a transient marker of small intestinal injury induced by luminal acetic acid and casein

Agents Actions. 1991 Sep;34(1-2):175-7. doi: 10.1007/BF01993270.

Abstract

We compared the time course of histamine release with other markers of intestinal injury in a rabbit model of necrotizing enterocolitis. Injury was induced by luminal acetic acid (200 mM) and casein (10 mg/ml) and experiments terminated after 45 min or 3 hr. Compared to saline controls there was a significant elevation of epithelial permeability (51Cr-EDTA clearance) and luminal protein levels at both time points. Luminal fluid histamine levels were approximately 120-fold greater than saline controls at 45 min but were indistinguishable from control values at 3 hr. We conclude that although mast cell activation is a characteristic of this model, elevations in histamine levels are transient.

MeSH terms

  • Acetates*
  • Acetic Acid
  • Animals
  • Biomarkers*
  • Caseins*
  • Epithelium / metabolism
  • Epithelium / pathology
  • Exudates and Transudates / metabolism
  • Histamine / analysis*
  • Intestinal Mucosa / metabolism
  • Intestinal Secretions / chemistry
  • Intestine, Small / pathology*
  • Male
  • Mast Cells / drug effects
  • Mast Cells / metabolism
  • Necrosis
  • Permeability
  • Proteins / metabolism
  • Rabbits

Substances

  • Acetates
  • Biomarkers
  • Caseins
  • Proteins
  • Histamine
  • Acetic Acid