Airway epithelial cell response to human metapneumovirus infection

Virology. 2007 Nov 10;368(1):91-101. doi: 10.1016/j.virol.2007.06.023. Epub 2007 Jul 25.

Abstract

Human metapneumovirus (hMPV) is a major cause of lower respiratory tract infections (LRTIs) in infants, elderly and immunocompromised patients. In this study, we show that hMPV can infect in a similar manner epithelial cells representative of different tracts of the airways. hMPV-induced expression of chemokines IL-8 and RANTES in primary small alveolar epithelial cells (SAE) and in a human alveolar type II-like epithelial cell line (A549) was similar, suggesting that A549 cells can be used as a model to study lower airway epithelial cell responses to hMPV infection. A549 secreted a variety of CXC and CC chemokines, cytokines and type I interferons, following hMPV infection. hMPV was also a strong inducer of transcription factors belonging to nuclear factor (NF)-kappaB, interferon regulatory factors (IRFs) and signal transducers and activators of transcription (STATs) families, which are known to orchestrate the expression of inflammatory and immunomodulatory mediators.

Publication types

  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Line
  • Chemokines / biosynthesis
  • Epithelial Cells / virology*
  • Humans
  • Interferon Regulatory Factors / metabolism
  • Interferon-Stimulated Gene Factor 3 / metabolism
  • Metapneumovirus / growth & development*
  • Metapneumovirus / immunology*
  • NF-kappa B / metabolism
  • Respiratory Mucosa / virology*

Substances

  • Chemokines
  • Interferon Regulatory Factors
  • Interferon-Stimulated Gene Factor 3
  • NF-kappa B