Predictive value of MSH2 gene expression in colorectal cancer treated with capecitabine

Clin Colorectal Cancer. 2007 Mar;6(6):433-5. doi: 10.3816/CCC.2007.n.012.

Abstract

Purpose: The objective of the present study was to evaluate the gene expression of the DNA mismatch repair gene MSH2 as a predictive marker in advanced colorectal cancer (CRC) treated with first-line capecitabine.

Patients and methods: Microdissection of paraffin-embedded tumor tissue, RNA extraction, and quantitative polymerase chain reaction were performed on tumors obtained from 37 patients with advanced CRC.

Results: The median relative gene expression of MSH2 was 0.65 (quartiles 0.5-0.8) in nonresponders and 1.25 (quartiles 0.92-1.38) for responders (P = 0.038). High expression of MSH2 was associated with a hazard ratio of 0.5 (95% confidence interval, 0.23-1.11; P = 0.083) in survival analysis.

Conclusion: The higher gene expression of MSH2 in responders and the trend for predicting overall survival indicates a predictive value of this marker in the treatment of advanced CRC with capecitabine.

Publication types

  • Clinical Trial, Phase II

MeSH terms

  • Aged
  • Antimetabolites, Antineoplastic / therapeutic use*
  • Biomarkers, Tumor*
  • Capecitabine
  • Colorectal Neoplasms / drug therapy
  • Colorectal Neoplasms / genetics*
  • Colorectal Neoplasms / mortality
  • DNA Mismatch Repair*
  • Deoxycytidine / analogs & derivatives*
  • Deoxycytidine / therapeutic use
  • Female
  • Fluorouracil / analogs & derivatives*
  • Fluorouracil / therapeutic use
  • Gene Expression*
  • Humans
  • Male
  • Microsatellite Instability / drug effects
  • Middle Aged
  • MutS Homolog 2 Protein* / deficiency
  • Prodrugs / therapeutic use*
  • Prognosis
  • Survival Analysis
  • Treatment Outcome*

Substances

  • Antimetabolites, Antineoplastic
  • Biomarkers, Tumor
  • Prodrugs
  • Deoxycytidine
  • Capecitabine
  • MSH2 protein, human
  • MutS Homolog 2 Protein
  • Fluorouracil