Perinatal alcohol exposure enhances nocistatin levels in adulthood

Addict Biol. 2007 Jun;12(2):173-5. doi: 10.1111/j.1369-1600.2007.00057.x.

Abstract

In earlier experiments perinatal hormonal imprinting by alcohol decreased the hormone content of immune cells for life. In the present study, both a single day (15% on the third postnatal day) and a long-term treatment schedule of alcohol exposure (3% for 21 days) of dams during lactation significantly (P < 0.01) enhanced endogenous levels of nocistatin in the blood plasma as well as in the cerebrospinal fluid of the offspring, measured in 3-month-old rats. Our data suggest that alcohol consumption during lactation can cause a life-long influence on nocistatin levels in the offspring and most likely modify nocistatin-related functions such as pain tolerance.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Age Factors
  • Alcoholism / physiopathology*
  • Animals
  • Animals, Newborn
  • Brain / drug effects
  • Dose-Response Relationship, Drug
  • Ethanol / toxicity*
  • Female
  • Imprinting, Psychological / drug effects*
  • Lactation*
  • Opioid Peptides / metabolism*
  • Pain Threshold / drug effects
  • Rats
  • Rats, Wistar

Substances

  • Opioid Peptides
  • nocistatin
  • Ethanol