Alpha1-adrenoceptors down-regulate ClC-2 chloride channels in epithelial cells from the acutely denervated jejunum

Eur J Pharmacol. 2007 Jun 22;565(1-3):202-6. doi: 10.1016/j.ejphar.2007.03.011. Epub 2007 Mar 24.

Abstract

Acute sympathetic denervation of the small intestine up-regulates alpha1-adrenoceptors on villus enterocytes and activation of these alpha1-adrenoceptors inhibits chloride secretion. We tested whether alpha1-adrenoceptor-mediated inhibition of chloride secretion was the result of reduced ClC-2 chloride channel expression. Phorbol myristate acetate (PMA) (a protein kinase C (PKC) activator) had no effect on ClC-2 levels. In contrast, alpha1-adrenoceptor activation significantly decreased ClC-2 protein levels in both the villus (1.58+/-0.19 to 0.75+/-0.19 arbitrary units) and crypt (1.69+/-0.15 to 0.37+/-0.23 arbitrary units) epithelial cells from the acutely denervated jejunum but not innervated controls. These data suggest that inhibition of chloride secretion following alpha1-adrenoceptor activation in the acutely denervated small intestine may be through ClC-2 down-regulation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Autonomic Denervation
  • CLC-2 Chloride Channels
  • Chloride Channels / analysis*
  • Down-Regulation
  • Intestinal Mucosa / chemistry*
  • Intestinal Mucosa / cytology
  • Jejunum / innervation*
  • Male
  • Phosphorylation
  • Protein Kinase C / physiology
  • Rats
  • Rats, Inbred Lew
  • Receptors, Adrenergic, alpha-1 / physiology*
  • Sympathetic Nervous System / physiology*

Substances

  • CLC-2 Chloride Channels
  • Chloride Channels
  • Receptors, Adrenergic, alpha-1
  • Protein Kinase C