A genetic family-based association study of OLIG2 in obsessive-compulsive disorder

Arch Gen Psychiatry. 2007 Feb;64(2):209-14. doi: 10.1001/archpsyc.64.2.209.

Abstract

Context: Obsessive-compulsive disorder (OCD) is a debilitating familial psychiatric illness with associated brain abnormalities in the white matter. The gene for oligodendrocyte lineage transcription factor 2 (OLIG2) is an essential regulator in the development of cells that produce white matter (myelin). The OLIG2 gene is also highly expressed in brain regions implicated in OCD.

Objectives: To examine OLIG2 as a candidate gene for OCD susceptibility and to explore whether comorbidity subtypes of OCD have distinct associations with OLIG2 and the functionally related OLIG1 gene. It was hypothesized a priori that OLIG2 and OLIG1 were associated with OCD regardless of the presence of comorbid Tourette disorder (TD), but not with TD alone.

Design: Family-based association candidate gene study.

Setting: Participants and their family members were recruited from tertiary care OCD and TD specialty clinics.

Participants: Families of 66 probands with OCD with and without TD and 31 probands with TD without OCD.

Main outcome measures: Genotypes of single nucleotide polymorphism markers and related haplotypes.

Results: The following 3 single nucleotide polymorphism markers on OLIG2 were associated with the OCD without TD phenotype: rs762178 (minor allele frequency, 35%; P<.001), rs1059004 (minor allele frequency, 44%; P = .005), and rs9653711 (minor allele frequency, 44%; P = .004). A 5-marker haplotype (A/C/T/T/G) constituting these single nucleotide polymorphisms and exonic single nucleotide polymorphisms rs6517137 and rs13046814 was undertransmitted (frequency, 32%; permuted P=.004), whereas the G/A/T/T/C haplotype (frequency, 22%; permuted P=.02) was overtransmitted to probands with OCD alone, with a significant global P value (permuted P=.008).

Conclusions: This is the first study reporting an association between OLIG2 and OCD, specifically when TD comorbidity is absent. The findings support a role for white matter abnormalities in the etiology of the disorder.

Publication types

  • Comparative Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Basic Helix-Loop-Helix Transcription Factors / genetics*
  • Chromosome Mapping
  • Comorbidity
  • Family*
  • Gene Frequency
  • Genetic Markers
  • Genetic Predisposition to Disease
  • Haplotypes / genetics
  • Humans
  • Linkage Disequilibrium
  • Nerve Tissue Proteins / genetics*
  • Obsessive-Compulsive Disorder / diagnosis
  • Obsessive-Compulsive Disorder / epidemiology
  • Obsessive-Compulsive Disorder / genetics*
  • Oligodendrocyte Transcription Factor 2
  • Pedigree
  • Phenotype
  • Polymorphism, Single Nucleotide / genetics
  • Tourette Syndrome / epidemiology
  • Tourette Syndrome / genetics

Substances

  • Basic Helix-Loop-Helix Transcription Factors
  • Genetic Markers
  • Nerve Tissue Proteins
  • OLIG1 protein, human
  • OLIG2 protein, human
  • Oligodendrocyte Transcription Factor 2