Cell-associated double-stranded RNA enhances antitumor activity through the production of type I IFN

J Immunol. 2006 Nov 1;177(9):6122-8. doi: 10.4049/jimmunol.177.9.6122.

Abstract

The efficacy of tumor cell vaccination largely depends on the maturation and activation status of the dendritic cell. Here we investigated the ability of soluble and tumor cell-associated dsRNA to serve as an adjuvant in the induction of protective adaptive antitumor responses. Our data showed that cell-associated dsRNA, but not soluble dsRNA, enhanced both tumor-specific CD8(+) and CD4(+) T cell responses. The cell-associated dsRNA increased the clonal burst of tumor-specific CD8(+) T cells and endowed them with an enhanced capacity for expansion upon a secondary encounter with tumor Ags, even when the CD8(+) T cells were primed in the absence of CD4(+) T cell help. The adjuvant effect of cell-associated dsRNA was fully dependent on the expression of TLR3 by the APCs and their subsequent production of type I IFNs, as the adjuvant effect of cell-associated dsRNA was completely abrogated in mice deficient in TLR3 or type I IFN signaling. Importantly, treatment with dsRNA-associated tumor cells increased the number of tumor-infiltrating lymphocytes and enhanced the survival of tumor-bearing mice. The data from our studies suggest that using cell-associated dsRNA as a tumor vaccine adjuvant may be a suitable strategy for enhancing vaccine efficacy for tumor cell therapy in cancer patients.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adjuvants, Immunologic / therapeutic use*
  • Animals
  • Antigen-Presenting Cells / drug effects
  • Antigen-Presenting Cells / immunology
  • CD4-Positive T-Lymphocytes / drug effects
  • CD4-Positive T-Lymphocytes / immunology
  • CD8-Positive T-Lymphocytes / drug effects
  • CD8-Positive T-Lymphocytes / immunology
  • Cancer Vaccines / therapeutic use*
  • Interferon Type I / biosynthesis*
  • Lymphocyte Depletion
  • Mice
  • Mice, Mutant Strains
  • RNA, Double-Stranded / therapeutic use*
  • Receptor, Interferon alpha-beta / genetics
  • Thymoma / drug therapy*
  • Thymoma / immunology
  • Thymus Neoplasms / drug therapy*
  • Thymus Neoplasms / immunology
  • Toll-Like Receptor 3 / genetics

Substances

  • Adjuvants, Immunologic
  • Cancer Vaccines
  • Interferon Type I
  • RNA, Double-Stranded
  • Toll-Like Receptor 3
  • Receptor, Interferon alpha-beta