Human cardiac sodium channels expressed in Xenopus oocytes

Am J Physiol. 1990 Mar;258(3 Pt 2):H903-6. doi: 10.1152/ajpheart.1990.258.3.H903.

Abstract

We report the expression of voltage-dependent Na+ channels in Xenopus oocytes injected with total RNA isolated from explanted human hearts. The expressed channels demonstrate characteristic voltage-dependent gating, inhibition by tetrodotoxin, and selectivity for Na+. Oocytes injected with sterile water or intentionally degraded RNA had no similar channel activity. The antiarrhythmic agent lidocaine (20 microM) inhibits current flow through the channel in a voltage-dependent fashion. Na+ channels expressed by injection of human cardiac RNA into Xenopus oocytes qualitatively resemble channels in the native tissue.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Electrophysiology
  • Gene Expression*
  • Homeostasis
  • Humans
  • Injections
  • Lidocaine / pharmacology
  • Myocardium / analysis
  • Myocardium / metabolism*
  • Oocytes / metabolism*
  • RNA / pharmacology
  • Rest
  • Sodium / pharmacology
  • Sodium Channels / drug effects
  • Sodium Channels / metabolism*
  • Sodium Channels / physiology
  • Xenopus laevis / metabolism*

Substances

  • Sodium Channels
  • RNA
  • Lidocaine
  • Sodium