ATP-activated channels in rat and bullfrog sensory neurons: concentration dependence and kinetics

J Neurosci. 1990 Jan;10(1):1-10. doi: 10.1523/JNEUROSCI.10-01-00001.1990.

Abstract

The concentration dependence and kinetics of ionic currents activated by extracellular adenosine 5'-triphosphate (ATP) were studied in voltage-clamped dorsal root ganglion neurons from rats and bullfrogs. About 40% of neurons of both species responded to ATP with an increase in membrane conductance. The ATP-activated currents were similar in the 2 species, except that currents in rat neurons desensitized faster. In bullfrog neurons, the conductance was half-maximally activated by about 3 microM ATP; at low concentrations, the conductance increased 3- to 7-fold for a doubling in [ATP], suggesting that several ATP molecules must bind in order to activate the current. A steeper concentration-response relationship than expected from 1:1 binding was also seen in rat neurons. The current activated quickly upon application of ATP and decayed quickly when ATP was removed. Activation kinetics were faster at higher [ATP], with time constants decreasing from about 200 msec at 0.3 microM ATP to about 10 msec at 100 microM ATP. Deactivation kinetics (tau approximately 100-200 msec) were independent of the ATP concentration. The rapid activation and deactivation make it seem likely that the ATP-activated current is mediated by direct ligand binding rather than by a second-messenger system. The experimental observations can be mimicked by a simple model in which ATP must bind to 3 identical, noninteracting sites in order to activate a channel. The potency and kinetics of ATP action were voltage-dependent, with hyperpolarization slowing deactivation and increasing ATP's potency. Deactivation kinetics were also sensitive to the concentration of external Ca, becoming faster in higher Ca.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Acetylcholine / pharmacology
  • Adenosine Triphosphate / pharmacology*
  • Animals
  • Calcium / pharmacology
  • Dose-Response Relationship, Drug
  • Electrophysiology
  • Ion Channels / drug effects
  • Ion Channels / physiology*
  • Kinetics
  • Neurons, Afferent / metabolism*
  • Osmolar Concentration
  • Rana catesbeiana
  • Rats

Substances

  • Ion Channels
  • Adenosine Triphosphate
  • Acetylcholine
  • Calcium