Tumor necrosis factor-alpha genetic polymorphism may contribute to progression of bovine leukemia virus-infection

Microbes Infect. 2006 Jul;8(8):2163-71. doi: 10.1016/j.micinf.2006.04.017. Epub 2006 Jun 2.

Abstract

In a previous report, we had indicated that in a sheep model, the expression of tumor necrosis factor (TNF)-alpha was closely associated with disease progression in sheep experimentally infected with bovine leukemia virus (BLV). However, individual variabilities are observed in these responses in BLV-infected animals. To attempt to identify genetic factors promoting the progression to BLV-induced lymphoma, we endeavored to determine whether there are any polymorphisms in the TNF-alpha gene among 291 individuals and whether this would affect the level of TNF-alpha expression and concomitant progression of BLV-induced disease or increase in the provirus load in the carriers. We found that the frequency of the TNF-alpha -824G allele, which has been associated with low transcription activity of the promoter/predicted enhancer region of the bovine TNF-alpha gene, was higher in individuals with BLV-induced lymphoma than in asymptomatic carrier individuals. In addition, we observed a tendency for increased BLV-provirus load in cattle with TNF-alpha -824G/G homozygote compared to TNF-alpha -824A/A homozygote or TNF-alpha -824A/G. These data suggest that the observed polymorphism in the promoter region of TNF-alpha gene could at least in part contribute to the progression of lymphoma in BLV-infection.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Base Sequence
  • Carrier State / virology
  • Cattle
  • Cell Line
  • Disease Progression
  • Enzootic Bovine Leukosis / genetics*
  • Enzootic Bovine Leukosis / immunology
  • Enzootic Bovine Leukosis / virology
  • Gene Expression
  • Gene Frequency
  • Leukemia Virus, Bovine
  • Lymphoma / genetics
  • Lymphoma / veterinary*
  • Lymphoma / virology
  • Molecular Sequence Data
  • Polymorphism, Genetic*
  • Promoter Regions, Genetic
  • Proviruses
  • RNA, Messenger / analysis
  • RNA, Messenger / genetics
  • Sequence Homology, Nucleic Acid
  • Transcriptional Activation
  • Tumor Necrosis Factor-alpha / genetics*
  • Tumor Necrosis Factor-alpha / immunology
  • Viral Load

Substances

  • RNA, Messenger
  • Tumor Necrosis Factor-alpha