Abstract
B cell development is characterized by a coordinated progression through defined stages that are controlled at several checkpoints. Signals from the pre-B cell receptor (pre-BCR) are essential for regulated transition from the pre-B cell stage. The adaptor protein SLP-65 plays a key role in this signaling pathway. Recent findings indicate an additional function of SLP-65 as a tumor suppressor that regulates pre-B cell proliferation. We will discuss here the potential mechanisms by which SLP-65 controls the pre-B cell checkpoint.
MeSH terms
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Adaptor Proteins, Signal Transducing / physiology*
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Agammaglobulinaemia Tyrosine Kinase
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Animals
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B-Lymphocytes / cytology*
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B-Lymphocytes / physiology
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Carrier Proteins / physiology*
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Cell Differentiation
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Cell Proliferation
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Humans
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Membrane Glycoproteins / metabolism
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Phosphoproteins / physiology*
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Pre-B Cell Receptors
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Precursor B-Cell Lymphoblastic Leukemia-Lymphoma / immunology
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Precursor B-Cell Lymphoblastic Leukemia-Lymphoma / physiopathology*
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Protein-Tyrosine Kinases
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Receptors, Antigen, B-Cell
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Signal Transduction*
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Tumor Suppressor Proteins / physiology*
Substances
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Adaptor Proteins, Signal Transducing
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B cell linker protein
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Carrier Proteins
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Membrane Glycoproteins
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Phosphoproteins
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Pre-B Cell Receptors
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Receptors, Antigen, B-Cell
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Tumor Suppressor Proteins
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Protein-Tyrosine Kinases
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Agammaglobulinaemia Tyrosine Kinase