Interleukin-10 promoter polymorphisms are associated with the mode and sequel of HBeAg seroconversion in patients with chronic hepatitis B virus infection

Liver Int. 2006 Apr;26(3):326-33. doi: 10.1111/j.1478-3231.2005.01241.x.

Abstract

Background: The influence of interleukin-10 (IL-10) gene promoter polymorphisms on the mode and sequel of HBeAg seroconversion (a favorable event usually) in patients with chronic Hepatitis B virus (HBV) infection has not been clarified.

Patients and methods: IL-10 genotyping and haplotype analyses of 340 HBsAg carriers and 100 volunteers with self-limiting HBV infection from southern China, a high prevalent area of HBV were performed according to the single nucleotide polymorphisms in its promoter (-1,082, -819 and -592) using a competitively differentiated PCR.

Results: High-producer genotype (GG at -1,082) or haplotype (GCC) was rarely found in patients from southern China (<1%). Intermediate-producer haplotype (ACC) was closely associated with chronic liver disease (P=0.004); compared with this, low-producer genotype (AA at -592) and haplotype (ATA) were closely associated with asymptomatic carriers (P=0.035 and 0.035). Intermediate-producer genotype (AC at -592) and haplotype (ACC) were closely associated with covert seroconversion of HBeAg (P=0.0086 and 0.0013) and progressive sequel after HBeAg seroconversion (P=0.013 and 0.0008), while, low-producer genotype (AA at -592) and haplotype (ATA) were closely associated with overt seroconversion of HBeAg (P=0.0023 and 0.0061) and silent sequel after HBeAg seroconversion (P=0.0009 and 0.001).

Conclusions: IL-10 gene promoter polymorphisms significantly influence the mode and sequel of HBeAg seroconversion in patients with chronic HBV infection.

Publication types

  • Comparative Study

MeSH terms

  • Adult
  • Biomarkers / blood
  • China / epidemiology
  • DNA Primers
  • DNA, Viral / genetics
  • Female
  • Genetic Predisposition to Disease
  • Genotype
  • Hepatitis B e Antigens / blood*
  • Hepatitis B, Chronic / genetics*
  • Hepatitis B, Chronic / immunology
  • Humans
  • Interleukin-10 / blood
  • Interleukin-10 / genetics*
  • Male
  • Middle Aged
  • Polymerase Chain Reaction
  • Polymorphism, Single Nucleotide*
  • Promoter Regions, Genetic*
  • Sequence Analysis, DNA

Substances

  • Biomarkers
  • DNA Primers
  • DNA, Viral
  • Hepatitis B e Antigens
  • Interleukin-10