Interleukin-2 (IL-2) induces tyrosine kinase-dependent translocation of active raf-1 from the IL-2 receptor into the cytosol

J Biol Chem. 1992 Aug 5;267(22):15281-4.

Abstract

Stimulation of the interleukin-2 (IL-2) receptor results in phosphorylation and activation of cytosolic Raf-1 serine/threonine kinase. Herein, we report that enzymatically active Raf-1 is physically associated with the IL-2 receptor beta chain (p75) in T-cell blasts. Following stimulation with IL-2, Raf-1 dissociates from the IL-2 receptor complex and translocates to the cytosol. Genistein, a protein tyrosine kinase inhibitor, prevents the dissociation of enzymatically active Raf-1 from the ligand-stimulated IL-2 receptor complex. These data favor a model of IL-2 receptor activation in which an IL-2-activated protein tyrosine kinase phosphorylates the IL-2 receptor and/or receptor-bound Raf-1. Following tyrosine phosphorylation, enzymatically active Raf-1 dissociates from the IL-2 receptor and translocates into the cytosol.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Blotting, Western
  • Cell Membrane / metabolism
  • Cells, Cultured
  • Cytosol / metabolism
  • Humans
  • Interleukin-2 / metabolism
  • Interleukin-2 / pharmacology*
  • Methionine / blood
  • Models, Biological
  • Monocytes / immunology
  • Monocytes / metabolism*
  • Protein Kinases / blood*
  • Protein-Tyrosine Kinases / blood*
  • Proto-Oncogene Proteins / blood*
  • Proto-Oncogene Proteins c-raf
  • Receptors, Interleukin-2 / metabolism*
  • Recombinant Proteins / metabolism
  • Recombinant Proteins / pharmacology

Substances

  • Interleukin-2
  • Proto-Oncogene Proteins
  • Receptors, Interleukin-2
  • Recombinant Proteins
  • Methionine
  • Protein Kinases
  • Protein-Tyrosine Kinases
  • Proto-Oncogene Proteins c-raf