N-acetylcysteine and S-methylcysteine inhibit MeIQx rat hepatocarcinogenesis in the post-initiation stage

Carcinogenesis. 2006 May;27(5):982-8. doi: 10.1093/carcin/bgi277. Epub 2005 Dec 7.

Abstract

N-acetylcysteine (NAC) and S-methylcysteine (SMC), water soluble organosulfur compounds contained in garlic, were evaluated for chemoprevention of hepatocarcinogenesis after 2-amino-3,8-dimethylimidazo[4,5-f]quinoxaline (MeIQx) initiation in rats. Intergastric treatment with NAC or SMC five times a week resulted in decreased numbers and areas of preneoplastic, glutathione S-transferase placental form (GST-P) positive foci of the liver in a dose-dependent manner. Moreover, cell proliferation was reduced in GST-P positive foci by NAC and SMC. Insulin-like growth factor I (IGF-I) and inducible nitric oxide synthase (iNOS) mRNA expressions were found downregulated in the liver by NAC. The studies indicate that NAC can serve as a chemopreventive agent for rat hepatocarcinogenesis induced by MeIQx by reducing cell proliferation, which may involve IGF-I and iNOS downregulation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acetylcysteine / pharmacology*
  • Animals
  • Anticarcinogenic Agents / pharmacology
  • Carcinogens*
  • Cell Proliferation
  • Cysteine / analogs & derivatives*
  • Cysteine / pharmacology
  • Down-Regulation
  • Glutathione Transferase / metabolism
  • Insulin-Like Growth Factor I / metabolism
  • Liver Neoplasms / chemically induced
  • Liver Neoplasms / metabolism*
  • Nitric Oxide Synthase Type II / metabolism
  • Placenta / metabolism
  • Quinoxalines*
  • Rats
  • Silver Staining
  • Time Factors

Substances

  • Anticarcinogenic Agents
  • Carcinogens
  • Quinoxalines
  • Insulin-Like Growth Factor I
  • 2-amino-3,8-dimethylimidazo(4,5-f)quinoxaline
  • S-methylcysteine
  • Nitric Oxide Synthase Type II
  • Glutathione Transferase
  • Cysteine
  • Acetylcysteine