[Effects of combined naloxone and methylprednisolone on nuclear factor-kappaB expression in the lung in acute lung injury in rats]

Zhongguo Wei Zhong Bing Ji Jiu Yi Xue. 2005 Jun;17(6):370-2.
[Article in Chinese]

Abstract

Objective: To investigate the effects of a combination of naloxone and methylprednisolone on nuclear factor-kappaB (NF-kappaB) p65 expression in the lung tissue in lipopolysaccharide (LPS)-induced acute lung injury (ALI) in rats.

Methods: ALI models were reproduced by intratracheal instillation of LPS (3 mg/kg). Four hours after LPS instillation, rats were randomly divided into five groups: normal saline group, LPS group, methylprednisolone group, naloxone group (LPS+naloxone) and combined drug group (LPS+naloxone+methylprednisolone). The level of interleukin-8 (IL-8) in serum was measured by immunoassay. Meanwhile, the expression of NF-kappaB p65 in the lung tissue was determined with immunohistochemical staining.

Results: Naloxone and methylprednisolone significantly reduced the LPS-induced increase in IL-8 concentrations in serum in vivo, and suppressed the activation of NF-kappaB p65 in the lung tissue.

Conclusion: NF-kappaB activation is involved in the LPS-induced ALI in rats. Combination of naloxone and methylprednisolone could suppress the increase of IL-8 content and NF-kappaB activation in the lung tissue of rat in vivo in our experiment.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute Lung Injury / chemically induced
  • Acute Lung Injury / metabolism*
  • Animals
  • Disease Models, Animal
  • Interleukin-8 / blood
  • Lipopolysaccharides / toxicity
  • Lung / drug effects
  • Lung / metabolism*
  • Male
  • Methylprednisolone / pharmacology*
  • NF-kappa B / metabolism*
  • Naloxone / pharmacology*
  • Random Allocation
  • Rats
  • Rats, Wistar

Substances

  • Interleukin-8
  • Lipopolysaccharides
  • NF-kappa B
  • Naloxone
  • Methylprednisolone