Abstract
Hyperhomocysteinemia represents an independent risk factor for atherothrombotic disease. Physiopathological mechanisms of accelerated progression of atherosclerosis in presence of hyperhomocysteinemia are complex. Herein we report a clinical case which emphasis the importance of screening elevated homocystein in the absence of conventional risk factors in patients who suffer from premature atherosclerosis.
MeSH terms
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Adult
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Coronary Artery Disease / blood*
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Coronary Artery Disease / diagnosis
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Coronary Artery Disease / genetics
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DNA Mutational Analysis
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Genetic Carrier Screening
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Homocysteine / blood*
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Humans
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Male
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Methylenetetrahydrofolate Reductase (NADPH2) / genetics
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Myocardial Infarction / blood*
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Myocardial Infarction / diagnosis
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Myocardial Infarction / genetics
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Protein S Deficiency / blood
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Protein S Deficiency / diagnosis
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Protein S Deficiency / genetics
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Prothrombin / genetics
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Recurrence
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Risk Factors
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Thrombophilia / blood
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Thrombophilia / diagnosis
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Thrombophilia / genetics
Substances
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Homocysteine
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Prothrombin
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Methylenetetrahydrofolate Reductase (NADPH2)