Older rhesus macaque infants are more susceptible to oral infection with simian-human immunodeficiency virus 89.6P than neonates

J Virol. 2005 Jan;79(2):1333-6. doi: 10.1128/JVI.79.2.1333-1336.2005.

Abstract

Earlier primate studies revealed that oral transmission of immunodeficiency viruses can occur at all ages [R. M. Ruprecht et al., J. Infect. Dis. 179(Suppl. 3):S408-S412, 1999]. Using a stock of pathogenic simian-human immunodeficiency virus, SHIV89.6P, we compared the 50% animal infectious dose needed to achieve systemic infection after oral challenge in newborn and older infant or juvenile rhesus macaques. Unexpectedly, the older monkeys required a 150-fold-lower virus challenge dose than the neonates (P=3.3 x 10(-5)). In addition, at least 60,000 times more virus was needed to achieve systemic infection in neonates by the oral route than by the intravenous route (P <1 x 10(-5)). Thus, route of inoculation and age are important determinants of SHIV89.6P infectivity in rhesus macaques.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Age Factors
  • Animals
  • Animals, Newborn
  • Macaca mulatta / virology*
  • Receptors, CCR5 / physiology
  • Receptors, CXCR4 / physiology
  • Simian Acquired Immunodeficiency Syndrome / etiology*
  • Simian Acquired Immunodeficiency Syndrome / virology
  • Viral Load

Substances

  • Receptors, CCR5
  • Receptors, CXCR4