Neurokinin-1 receptor interacts with PrP(106-126)-induced dendritic cell migration and maturation

J Neuroimmunol. 2005 Jan;158(1-2):153-8. doi: 10.1016/j.jneuroim.2004.09.003.

Abstract

Circulating monocytic cells may mediate neuroinvasion in transmissible spongiform encephalopathies by transporting prion protein (PrP) from sites of entry to the nervous system. Factors regulating monocyte-derived dendritic cell (DC) functions in neuronal tissue include neurogenic mediators, but their interactions with prion-infected DCs are unknown. Here, we report that neuropeptides regulate the interaction of DCs exposed to PrP(106-126). Inhibition of neurokinin-1-receptors activation with neurokinin-1-receptor antagonist or antibody attenuates substance P-induced enhancement of DC migration induced by PrP(106-126) and prion-protein-induced DC maturation. Other neuropeptides arrest chemotaxis. Data support a priming role of the neuropeptide substance P in PrP(106-126)-induced migration of DCs involving neurokinin-1-receptors.

Publication types

  • Comparative Study

MeSH terms

  • Antibodies / pharmacology
  • B7-1 Antigen / metabolism
  • Cell Movement / drug effects*
  • Cells, Cultured
  • Cellular Senescence / drug effects*
  • Cellular Senescence / physiology
  • Chemotaxis / drug effects*
  • Chemotaxis / physiology
  • Dendritic Cells / drug effects*
  • Dose-Response Relationship, Drug
  • Drug Interactions
  • Flow Cytometry / methods
  • HLA-DR Antigens / metabolism
  • Humans
  • Neurokinin-1 Receptor Antagonists
  • Neuropeptides / pharmacology
  • Peptide Fragments / pharmacology*
  • Prions / pharmacology*
  • Receptors, Neurokinin-1 / immunology
  • Receptors, Neurokinin-1 / metabolism*
  • Statistics, Nonparametric

Substances

  • Antibodies
  • B7-1 Antigen
  • HLA-DR Antigens
  • Neurokinin-1 Receptor Antagonists
  • Neuropeptides
  • Peptide Fragments
  • Prions
  • Receptors, Neurokinin-1
  • prion protein (106-126)