Activation of the cytokine network and unfavorable outcome in patients with yellow fever

J Infect Dis. 2004 Nov 15;190(10):1821-7. doi: 10.1086/425016. Epub 2004 Oct 12.

Abstract

To study the contribution of inflammatory mediators to the pathogenesis of yellow fever (YF), the serum levels of several cytokines and chemokines were measured in 7 patients with fatal YF (f-YF), 11 patients with nonfatal hemorrhagic YF (nf/h-YF), and 18 patients with nonfatal nonhemorrhagic YF (nf/nh-YF). The levels of interleukin (IL)-6, monocyte chemoattractant protein-1, interferon-inducible protein (IP)-10, tumor necrosis factor- alpha , and IL-1 receptor antagonist (IL-1RA) were all statistically significantly higher in the patients with f-YF than in those with nf/nh-YF. In patients with nf/h-YF, only levels of IP-10 and IL-1RA were significantly elevated. The high levels of pro- and anti-inflammatory cytokines and chemokines in serum from patients with f-YF are reminiscent of those seen in patients with bacterial sepsis. This finding has implications for the understanding of the pathophysiology of YF and the development of therapeutic strategies.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adolescent
  • Adult
  • Aged
  • Chemokine CCL2 / blood
  • Chemokine CXCL10
  • Chemokines, CXC / blood
  • Child
  • Child, Preschool
  • Cytokines / blood*
  • Cytokines / immunology
  • Female
  • Guinea
  • Hemorrhage
  • Humans
  • Inflammation Mediators / blood*
  • Inflammation Mediators / immunology
  • Interleukin 1 Receptor Antagonist Protein
  • Interleukin-6 / blood
  • Male
  • Middle Aged
  • Sialoglycoproteins / blood
  • Tumor Necrosis Factor-alpha / analysis
  • Yellow Fever / immunology*
  • Yellow Fever / pathology
  • Yellow Fever / physiopathology

Substances

  • CCL2 protein, human
  • Chemokine CCL2
  • Chemokine CXCL10
  • Chemokines, CXC
  • Cytokines
  • IL1RN protein, human
  • Inflammation Mediators
  • Interleukin 1 Receptor Antagonist Protein
  • Interleukin-6
  • Sialoglycoproteins
  • Tumor Necrosis Factor-alpha