Cell type-specific tyrosine phosphorylation of IL-2 receptor beta chain in response to IL-2

FEBS Lett. 1992 Sep 21;310(1):22-6. doi: 10.1016/0014-5793(92)81137-b.

Abstract

Functional activities of the IL-2 receptor (IL-2R) beta chain exogenously expressed on lymphoid and non-lymphoid cells were examined in terms of phosphorylation of IL-2R beta and cell growth. Lymphoid MOLT-4 and its transfectants expressing IL-2R beta either alone or with IL-2R alpha chain were found to be rapidly phosphorylated predominantly at tyrosine residues of IL-2R beta and to be affected in their growth in an IL-2-dependent manner. In contrast, IL-2 induced neither phosphorylation of IL-2R beta nor cell growth in non-lymphoid transfectants derived from COS7, HeLa and L929, even though they acquired the IL-2 binding ability when coexpressed as IL-2R beta and IL-2R alpha. These results suggest that IL-2 induces activation of a tyrosine kinase possibly associated with IL-2R beta in a cell type-specific manner.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Cell Division / drug effects
  • Cell Line
  • Electrophoresis, Gel, Two-Dimensional
  • Humans
  • Interleukin-2 / metabolism*
  • Interleukin-2 / pharmacology
  • Phosphorylation
  • Precipitin Tests
  • Receptors, Interleukin-2 / metabolism*
  • Thymidine / metabolism
  • Transfection
  • Tumor Cells, Cultured
  • Tyrosine / metabolism*

Substances

  • Interleukin-2
  • Receptors, Interleukin-2
  • Tyrosine
  • Thymidine