Transcription factor GATA-4 is a marker of anaplasia in adrenocortical neoplasms of the domestic ferret (Mustela putorius furo)

Vet Pathol. 2004 Jul;41(4):446-9. doi: 10.1354/vp.41-4-446.

Abstract

Adrenocortical neoplasms are a common cause of morbidity in neutered ferrets. Recently we showed that gonadectomized DBA/2J mice develop adrenocortical tumors that express transcription factor GATA-4. Therefore, we screened archival specimens of adrenocortical neoplasms from neutered ferrets to determine whether GATA-4 could be used as a tumor marker in this species. Nuclear immunoreactivity for GATA-4 was evident in 19/22 (86%) of ferret adrenocortical carcinomas and was prominent in areas exhibiting myxoid differentiation. Normal adrenocortical cells lacked GATA-4 expression. Two other markers of adrenocortical tumors in gonadectomized mice, inhibin-alpha and luteinizing hormone receptor, were coexpressed with GATA-4 in some of the ferret tumors. No GATA-4 expression was observed in three cases of nodular hyperplasia, but patches of anaplastic cells expressing GATA-4 were evident in 7/14 (50%) of tumors classified as adenomas. We conclude that GATA-4 can function as a marker of anaplasia in ferret adrenocortical tumors.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Adrenal Cortex Neoplasms / metabolism
  • Adrenal Cortex Neoplasms / pathology
  • Adrenal Cortex Neoplasms / veterinary*
  • Adrenocortical Carcinoma / metabolism
  • Adrenocortical Carcinoma / pathology
  • Adrenocortical Carcinoma / veterinary*
  • Animals
  • Biomarkers, Tumor / metabolism*
  • DNA-Binding Proteins / metabolism*
  • Ferrets*
  • GATA4 Transcription Factor
  • Immunohistochemistry / veterinary
  • Inhibins / metabolism
  • Receptors, LH / metabolism
  • Transcription Factors / metabolism*

Substances

  • Biomarkers, Tumor
  • DNA-Binding Proteins
  • GATA4 Transcription Factor
  • Receptors, LH
  • Transcription Factors
  • inhibin-alpha subunit
  • Inhibins