Inhibitory effects of KAT-681, a liver-selective thyromimetic, on development of hepatocellular proliferative lesions in rats induced by 2-acetylaminofluorene and partial hepatectomy after diethylnitrosamine initiation

Arch Toxicol. 2004 Aug;78(8):460-6. doi: 10.1007/s00204-004-0561-4. Epub 2004 Mar 19.

Abstract

To examine the potential inhibitory effects of a novel liver-selective thyromimetic, KAT-681 (KAT), on the development of hepatocellular proliferative lesions, male F344 rats were given a single intraperitoneal injection of 150 mg/kg diethylnitrosamine (DEN), followed by gavage administration of 7.5 mg/kg per day of 2-acetylaminofluorene (2-AAF) twice daily from weeks 2 to 4 with partial hepatectomy (PH) at week 3. From 5 weeks after the completion of 2-AAF administration, the rats were orally dosed with 0.04, 0.1, or 0.25 mg/kg per day KAT for 3 weeks, and subjected to morphometric analysis of the induced glutathione S-transferase placental form (GST-P)-positive lesions and hepatocellular adenomas (HCAs). Administration of KAT significantly and dose-dependently reduced the total area of GST-P-positive lesions (by 34-48%) and also their numbers (by 20-44%), their mean size not being significantly changed. No effects on the number of HCAs were apparent, although a reduction in their mean size was detected at a dose of 0.25 mg/kg per day KAT (by 34%). On biochemical analysis, serum activity of gamma-glutamyl transpeptidase, an enzyme related to hepatocarcinogenesis, was markedly reduced in rats given 0.25 mg/kg per day KAT (by 64%). The results of the present study thus suggest that KAT inhibits the development of altered hepatocellular foci and might be a promising chemopreventive agent for hepatocarcinogenesis.

MeSH terms

  • 2-Acetylaminofluorene / toxicity*
  • Animals
  • Apoptosis / drug effects
  • Body Weight / drug effects
  • Carcinogens / toxicity*
  • Cell Proliferation / drug effects
  • Diethylnitrosamine / toxicity*
  • Eating / drug effects
  • Glutathione Transferase / metabolism
  • Hepatectomy
  • Hepatocytes / drug effects*
  • Hydrogen-Ion Concentration
  • Immunohistochemistry
  • Liver / drug effects*
  • Liver / pathology
  • Male
  • Malonates / pharmacology*
  • Organ Size / drug effects
  • Phenyl Ethers / pharmacology*
  • Proliferating Cell Nuclear Antigen / metabolism
  • Rats
  • Rats, Inbred F344
  • Thyroid Gland / drug effects*

Substances

  • Carcinogens
  • Malonates
  • N-(4-(3-((4-fluorophenyl)hydroxymethyl)-4-hydroxyphenoxy)-3,5-dimethylphenyl) malonamic acid sodium
  • Phenyl Ethers
  • Proliferating Cell Nuclear Antigen
  • Diethylnitrosamine
  • 2-Acetylaminofluorene
  • Glutathione Transferase