Neutrophils are stimulated by syncytiotrophoblast microvillous membranes to generate superoxide radicals in women with preeclampsia

Am J Obstet Gynecol. 2004 Jan;190(1):252-8. doi: 10.1016/j.ajog.2003.07.003.

Abstract

Objective: This study was undertaken to determine whether syncytiotrophoblast microvillous membranes (STBMs) stimulate maternal neutrophils to produce superoxide radicals in women with preeclampsia.

Study design: Serum levels of tissue polypeptide antigen (TPA), which is a marker for STBM, were measured in 25 nulliparous women (10 with mild preeclampsia, 6 with severe preeclampsia, and 9 controls). Superoxide production by maternal neutrophils from cases and controls and by donor neutrophils cocultured with the STBMs from cases and controls was measured spectrophotometrically by reduction of ferricytochrome C.

Results: Maternal TPA levels were significantly greater among cases than controls (P=.005). Superoxide production by maternal neutrophils and donor neutrophils cultured with STBM from cases of preeclampsia was greater than controls (P values.006 and.019, respectively), and dose-response relationships were observed. Superoxide production by maternal leukocytes was correlated with superoxide induction by STBMs in culture (P=.007).

Conclusion: STBMs in maternal blood induce neutrophils to generate superoxide radicals that may cause endothelial dysfunction in women with preeclampsia.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Case-Control Studies
  • Chorionic Villi / physiopathology*
  • Coculture Techniques
  • Female
  • Humans
  • Membranes / physiopathology
  • Neutrophils / metabolism*
  • Pre-Eclampsia / pathology
  • Pre-Eclampsia / physiopathology*
  • Pregnancy
  • Superoxides / metabolism*
  • Tissue Donors
  • Tissue Polypeptide Antigen / blood
  • Trophoblasts*

Substances

  • Tissue Polypeptide Antigen
  • Superoxides