Intrathecal clonidine and severe hypotension after cardiopulmonary bypass

Anesth Analg. 2003 Nov;97(5):1251-1253. doi: 10.1213/01.ANE.0000083526.08033.20.

Abstract

The use of intrathecal clonidine as an adjunct for the management of chronic pain, intra- and postoperative analgesia is gaining an increase in popularity. However, antinociceptive doses of intrathecal clonidine may produce pronounced hemodynamic side effects, including hypotension and bradycardia. In this report, we present a case of severe hypotension after cardiopulmonary bypass in a patient with intrathecal clonidine infusion. We postulate that the intrathecally administered alpha 2-agonist clonidine reduced our patient's ability to tolerate the hemodynamic lability that is present during the separation from cardiopulmonary bypass by potentially inhibiting sympathetic nervous system activity, renin-angiotensin system, or vasopressin release. The authors report a case of severe hypotension after cardiopulmonary bypass in a patient receiving intrathecal clonidine infusion for chronic neuropathic pain.

Publication types

  • Case Reports

MeSH terms

  • Adrenergic alpha-Agonists / adverse effects*
  • Adrenergic alpha-Agonists / therapeutic use
  • Cardiopulmonary Bypass*
  • Clonidine / adverse effects*
  • Clonidine / therapeutic use
  • Hemodynamics / drug effects
  • Humans
  • Hypotension / chemically induced*
  • Injections, Spinal
  • Male
  • Middle Aged
  • Pain, Postoperative / drug therapy
  • Postoperative Complications / chemically induced*

Substances

  • Adrenergic alpha-Agonists
  • Clonidine