Therapeutic effect of propofol in the treatment of endotoxin-induced shock in rats

J Huazhong Univ Sci Technolog Med Sci. 2003;23(3):320-3. doi: 10.1007/BF02829527.

Abstract

To assess the potential therapeutic effect of propofol in the treatment of endotoxemia, 76 rats were randomly assigned to 5 groups: control group(A), endotoxemic group(B), pre-treatment group(C), simultaneous treatment group(D) and post-treatment group(E). Five h after endotoxin injection, PO2, pH, MAP, plasma concentrations of Nitrite/nitrate (NO2-/NO3-) and mortality rates were assessed in each group. After the rats were sacrificed, lung tissue was sampled to measure myeloperoxidase (MPO) activity and tumor necrosis factor (TNF)-alpha contents. It was found that endotoxin injection produced progressive hypotension, metabolic acidosis, and a large increase in the plasma NO2-/NO3- concentrations and increased mortality rates in 5 h. Endotoxin injection significantly increased MPO activity and TNF-alpha contents in lung tissue (P < 0.01 or P < 0.05). These changes response to endotoxin were significantly attenuated in the groups B, C and D. But these beneficial effects were blunted in the group E. The results suggest that propofol administration may offer advantages in endotoxemia.

MeSH terms

  • Animals
  • Free Radical Scavengers / therapeutic use*
  • Lipopolysaccharides
  • Lung / metabolism
  • Male
  • Nitric Oxide / blood
  • Peroxidase / metabolism*
  • Propofol / therapeutic use*
  • Random Allocation
  • Rats
  • Rats, Wistar
  • Shock, Septic / chemically induced
  • Shock, Septic / drug therapy*
  • Tumor Necrosis Factor-alpha / metabolism*

Substances

  • Free Radical Scavengers
  • Lipopolysaccharides
  • Tumor Necrosis Factor-alpha
  • Nitric Oxide
  • Peroxidase
  • Propofol